Sudden Cardiac Arrest & Cardiopulmonary Resuscitation

This section on Sudden Cardiac Arrest and Resuscitation delves into the many aspects of sudden cardiac arrest. It is a comprehensive resource aimed at clinicians, from paramedics to in-hospital personnel. It covers the pathophysiology, clinical context, and treatments involved in resuscitation, complete with self-assessment tests for each topic explored.

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Introduction to cardiac arrest

Hearts too good to die In 1961 the Journal of the American Medical Association (JAMA) published an article titled Hearts too good to die (JAMA,

Epidemiological aspects of cardiac arrest

Data sources for monitoring cardiac arrest and resuscitation There are currently several large registries monitoring resuscitation practices. Some data sources also monitor sudden cardiac arrest.

The 3-phase model of sudden cardiac arrest

The course from sudden cardiac arrest (SCA) to death follows a pathophysiological process that affects the effectiveness of the interventions. Numerous studies demonstrate that the

Sudden cardiac arrest and resuscitation

The physiology of cardiopulmonary resuscitation (CPR)

The forces that drive coronary and cerebral perfusion, and ventilation of the lungs during cardiopulmonary resuscitation (CPR) differ substantially from normal circumstances. The purpose of

Risk factors for sudden cardiac arrest

Predicting sudden cardiac arrest The purpose of discovering and measuring risk factors is to obtain estimates of risk, tailor management according to the risk and

Causes of sudden cardiac arrest

It is commonly stated that approximately 80% of all cardiac arrests are caused by acute or chronic coronary artery disease, with the latter being the

Arrhythmias before and during cardiac arrest

Cardiac arrest is a result of tachyarrhythmia, bradyarrhythmia or asystole. The tachyarrhythmias include ventricular fibrillation (VF) and pulseless ventricular tachycardia (VT). The bradyarrhythmias include all

Cardiac arrest due to electrolyte imbalance

Electrolyte imbalance and cardiac arrest Significant electrolyte imbalances can precipitate cardiac arrest and are potentially reversible etiologies when promptly and appropriately addressed. Both venous and

Cardiac arrest due to anaphylaxis

Anaphylactic cardiac arrest Anaphylactic cardiac arrest is managed following the cardiopulmonary resuscitation (CPR) algorithm, incorporating an intravenous administration of 1 mg epinephrine. Predominant causes of

Cardiac arrest due to hypoxia and asphyxia

Hypoxia and asphyxiation Etiologies of hypoxic cardiac arrest: The natural course of asphyxia Should ventilation be entirely inhibited (as seen in acute airway obstructions), a

Traumatic cardiac arrest

Cardiac arrest due to trauma In cases of traumatic cardiac arrest, addressing reversible etiologies takes precedence over chest compressions. If trauma is not definitively established

Cardiac tamponade causing cardiac arrest

Tamponade and cardiac arrest Cardiac arrest associated with tamponade has an exceptionally poor prognosis. In the vast majority of these cases, the tamponade develops acutely

Cardiac arrest due to pneumothorax

Pneumothorax Pneumothorax is characterized by the presence of free air in the thoracic cavity. While this can occasionally be benign and manifest even in healthy

Cardiac arrest during sepsis

Sepsis-induced cardiac arrest Sepsis is a life-threatening condition characterized by systemic inflammatory response syndrome (SIRS), disseminated intravascular coagulation (DIC), and hypoperfusion manifesting as hypotension. The

Sudden cardiac arrest: Case 1

Each recording presents two leads recorded simultaneously. Sex: Female Age: 72 years History: heart failure Medications: digoxin, quinidine

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Wiggers diagram showing pressure, volume, Doppler signal, ECG and AV valves during the cardiac cycle. Doppler recording of mitral valve flow during diastole. (a) = active atrial filling; (b) = increased atrial pressure due to bulging of mitral valve into the left atrium, when valve closes; (c) = passive atrial filling.